skull fracture indicates substantial force was involved
basilar skull fractures have a high incidence of intracranial haemorrhage
the following assumes other injuries have been managed and a primary and secondary survey carries out, in particular, cervical spine injury should be actively considered, as up to 15% of patients with skull fracture sustain a Cx spine fracture as well.
clinical features
Battle sign:
retroauricular or mastoid bruising - usually occurs 1-3 days after injury
Racoon eyes:
bilateral periorbital haematoma - usually occurs 1-3 days after injury
haemotympanum:
blood behind the ear drum suggests fracture of petrous ridge of the temporal bone and tends to occur within hours of injury
CSF leak:
dural tears may allow CSF to leak into the paranasal sinuses and then into the nose, or into the middle ear.
CSF leaks occur in up to 45% of basilar fractures with onset of up to several days post injury
these may manifest either by clear rhinorrhoea or clear otorrhoea
testing rhinorrhoea for glucose can give false positives and negatives and not clinically useful
nasal secretions and tears may have reducing substances giving false positives
hyperglycaemia may give false positive
meningitis may lower CSF glucose giving false negative
subconjunctival haemorrhage without visible posterior border:
subconjunctival haemorrhage is a common condition, but in the setting of head trauma is suggestive of basilar fracture if the posterior border of the haemorrhage cannot be seen.
complications
extradural haematoma:
as the most common basilar fracture involves the temporal bone, and thus near the middle meningeal artery, extradural haemorrhage is a significant risk
subdural haematoma:
a significant risk, particular if patients are on anticoagulants
persistent CSF leak:
most resolve within 1 week without complication
those with persistent leak > 1 week should be given prophylactic antibiotics as per penetrating head trauma
CNS infection:
meningitis risk from CSF leaks has been estimated at 3% in the 1st week and rises substantially in those with persistent CSF leak > 1 week
cranial nerve palsy:
acute onset facial nerve palsy at time of injury indicates nerve transection
delayed onset nerve palsy suggests neuropraxia from compression or contusion
traumatic carotid cavernous fistula (TCCF):
a rare complication occurring in ~4% of patients with basilar fractures
carotid artery thrombosis or dissection:
a rare complication
Mx in ED of the patient with an isolated basilar skull fracture
CT brain to exclude intracranial haemorrhage if:
evidence or suspicion of basilar skull fracture, or,
patient on anticoagulants with a head injury, or,
other trauma indicators for performing CT brain such as persistent vomiting, reduced GCS, intoxication, etc.
patients for active Rx who are at increased risk of bleeding (eg. on anticoagulants such as warfarin or clopidogrel):
reverse warfarin
should be admitted and observed for at least 24 hours with hourly neuro obs at least for the first 6 hours.
repeat CT brain if subsequent deterioration, as there is a risk of delayed subdural haematoma collecting.
patients with CSF leak > 1 week should be given prophylactic antibiotics1)