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allergy [2026/09/23 12:43] – [allergy and hypersensitivity reactions] gary1allergy [2026/09/23 12:43] (current) – [Introduction] gary1
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   *many people have allergies which may be to medicines, biological substances such as from bee stings, components in foods, contact substances or inhaled in the air   *many people have allergies which may be to medicines, biological substances such as from bee stings, components in foods, contact substances or inhaled in the air
   *traditionally the various types of allergic reactions have been classified under the Gell and Coombs hypersensitivity reactions (see below)   *traditionally the various types of allergic reactions have been classified under the Gell and Coombs hypersensitivity reactions (see below)
-  *recently it has also been proposed that sensory nerves may "remember" allergens and also become a source of initiating reactions on subsequent exposures+  *recently it has also been proposed that sensory nerves may "remember" protease allergens and also become a source of initiating reactions on subsequent exposures
     * protease allergen exposure induced sustained mechanistic target of rapamycin complex 1 (mTORC1) kinase signaling and transcriptional activator peroxisome proliferator-activated receptor gamma coactivator-1α (PGC-1α)-associated mitochondrial remodeling in sensory neurons, establishing a metabolically primed state with enhanced neuronal responsiveness. Upon allergen re-exposure, this state drove enhanced itch, CD301b+ dendritic cell migration, and Th2 cell differentiation(([[https://www.cell.com/immunity/fulltext/S1074-7613(26)00349-3|2026: Sensory neuronal mTORC1 kinase signaling establishes neuroimmune memory that initiates allergic immunity]]))     * protease allergen exposure induced sustained mechanistic target of rapamycin complex 1 (mTORC1) kinase signaling and transcriptional activator peroxisome proliferator-activated receptor gamma coactivator-1α (PGC-1α)-associated mitochondrial remodeling in sensory neurons, establishing a metabolically primed state with enhanced neuronal responsiveness. Upon allergen re-exposure, this state drove enhanced itch, CD301b+ dendritic cell migration, and Th2 cell differentiation(([[https://www.cell.com/immunity/fulltext/S1074-7613(26)00349-3|2026: Sensory neuronal mTORC1 kinase signaling establishes neuroimmune memory that initiates allergic immunity]]))
 =====Hypersensitivity reactions - the Gell & Coombs classification===== =====Hypersensitivity reactions - the Gell & Coombs classification=====
allergy.txt · Last modified: 2026/09/23 12:43 by gary1

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