Table of Contents

Mx of psychostimulant toxicity from amphetamines and related substances

see also:

do not use beta blockers (may result in uncontrolled hypertension) or phenytoin (potential cardiac adverse effects)

Clinical manifestations of psychostimulant toxicity

acute toxicity

cardiovascular
CNS
other sympathetic
pulmonary
GIT
metabolic
other

chronic toxicity

Clinical signs indicating significant amphetamine toxicity

DDx of amphetamine toxicity

other drug toxicity

systemic conditions

Specific Mx interventions in toxicity

agitation

  • treat aggressively aiming for a state of rousable drowsiness without causing respiratory depression
  • no sedation protocol is 100% safe, and thus these are only indicated when all other simpler, safer measures fail or are inappropriate, and the patient is deemed a significant risk to self or others. It is a crisis management tool.
  • it is critical in the initial period of physical restraint and parenteral sedation for direct visual observation of the patient's cardio-respiratory status to be maintained, and once the patient has settled sufficiently, electronic monitoring should be initiated as soon as reasonably possible to ensure patient safety.
  • a medical officer with advanced airway skills should remain with the patient whilst aggressive sedative Rx occurs
  • observations of vital signs and patient status should occur continuously in 1st 10min after parenteral sedation and every 10min for 1st 30min, then every 15min for 60min, then hourly for 4 hours after last dose or until awake
  • if resp. depression does occur following benzodiazepine sedation, only consider using flumazenil to reverse it if there is no evidence of concomitant pro-arrhythmic or pro-convulsant drug taken such as tricyclic antidepressants, or they are a regular benzodiazepine user, otherwise there is risk of seizures or cardiac arrest - usually safer to intubate than give flumazenil as inaccuracy of drug use history and polypharmacy tend to be issues.
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seizures

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decreased conscious state

hyponatraemia

hyperthermia

rhabdomyolysis

hypertension

SVT

VT/VF

ischaemic chest pain

cerebrovascular emergencies

References