fracture_skull_basilar
Table of Contents
fracture base of skull
introduction
- skull fracture indicates substantial force was involved
- basilar skull fractures have a high incidence of intracranial haemorrhage
- the following assumes other injuries have been managed and a primary and secondary survey carries out, in particular, cervical spine injury should be actively considered, as up to 15% of patients with skull fracture sustain a Cx spine fracture as well.
clinical features
- Battle sign:
- retroauricular or mastoid bruising - usually occurs 1-3 days after injury
- Racoon eyes:
- bilateral periorbital haematoma - usually occurs 1-3 days after injury
- haemotympanum:
- blood behind the ear drum suggests fracture of petrous ridge of the temporal bone and tends to occur within hours of injury
- CSF leak:
- dural tears may allow CSF to leak into the paranasal sinuses and then into the nose, or into the middle ear.
- CSF leaks occur in up to 45% of basilar fractures with onset of up to several days post injury
- these may manifest either by clear rhinorrhoea or clear otorrhoea
- testing rhinorrhoea for glucose can give false positives and negatives and not clinically useful
- nasal secretions and tears may have reducing substances giving false positives
- hyperglycaemia may give false positive
- meningitis may lower CSF glucose giving false negative
- subconjunctival haemorrhage without visible posterior border:
- subconjunctival haemorrhage is a common condition, but in the setting of head trauma is suggestive of basilar fracture if the posterior border of the haemorrhage cannot be seen.
complications
- extradural haematoma:
- as the most common basilar fracture involves the temporal bone, and thus near the middle meningeal artery, extradural haemorrhage is a significant risk
- subdural haematoma:
- a significant risk, particular if patients are on anticoagulants
- persistent CSF leak:
- most resolve within 1 week without complication
- those with persistent leak > 1 week should be given prophylactic antibiotics as per penetrating head trauma
- CNS infection:
- meningitis risk from CSF leaks has been estimated at 3% in the 1st week and rises substantially in those with persistent CSF leak > 1 week
- cranial nerve palsy:
- acute onset facial nerve palsy at time of injury indicates nerve transection
- delayed onset nerve palsy suggests neuropraxia from compression or contusion
- traumatic carotid cavernous fistula (TCCF):
- a rare complication occurring in ~4% of patients with basilar fractures
- carotid artery thrombosis or dissection:
- a rare complication
Mx in ED of the patient with an isolated basilar skull fracture
- CT brain to exclude intracranial haemorrhage if:
- evidence or suspicion of basilar skull fracture, or,
- patient on anticoagulants with a head injury, or,
- other trauma indicators for performing CT brain such as persistent vomiting, reduced GCS, intoxication, etc.
- patients for active Rx who are at increased risk of bleeding (eg. on anticoagulants such as warfarin or clopidogrel):
- reverse warfarin
- should be admitted and observed for at least 24 hours with hourly neuro obs at least for the first 6 hours.
- repeat CT brain if subsequent deterioration, as there is a risk of delayed subdural haematoma collecting.
- patients with CSF leak > 1 week should be given prophylactic antibiotics1)
fracture_skull_basilar.txt · Last modified: 2017/05/30 06:16 by 127.0.0.1