an active pre-existing co-morbidity may have been the cause of the trauma (eg. cardiac syncope, occult GIT blood loss, acute stroke, leaking AAA) and these may easily be not assessed with attention focusing on the injuries
reduced pulmonary reserves
lower vital capacity, functional residual capacity and FEV1
thus less able to compensate for metabolic disturbances
reduced cardiovascular reserves
reduced ability to raise cardiac output
less sensitive to catecholamines
current medications may increase risks:
anticoagulants increase risk of bleeding, including increased subdural haematoma risk (CHI on warfarin increases risk of intracranial bleed to around 15%! - CT all such patients!)
other risk factors for worse outcomes from the trauma itself:
cerebrovascular regulation decreased, increasing risk of secondary brain injury during hypotension periods
central cord syndrome from Cx spine injury is more common in the elderly, especially in those with cervical spondylosis who sustain an hyperextension injury (results in motor impairment UL > LL; bladder dysfunction; variable degree of sensory loss below level)
more difficult to assess injuries as patient may be less cooperative, less aware, diminished pain sensation, lax abdominal wall musculature or pre-existing conditions impact assessment - eg. Cx spine injury, blunt abdo trauma is difficult to assess clinically.
more likely to have an adverse medication or infusion event
increased Vd of a drug and impaired elimination
increased risk of delirium and falls while in hospital and upon discharge
higher risk of APO following rapid infusions - consider 500mL boluses instead of 1L, and start early blood transfusion to minimise crystalloid volumes
higher risk of over-sedation and respiratory depression
more likely to have complications from prolonged spinal immobilisation
neck pain and injury
airway compromise
aspiration / impaired respiration
decubitus ulcers
urinary retention
More likely to be under-triaged and more difficult to detect the deteriorating patient
more likely to remain within normal RR range despite developing hypoxia or hypercapnia
need more aggressive monitoring such as oxygen saturation and end tidal CO2 if respiratory issues or requiring high levels of analgesics
less likely to mount a tachycardia response to hypovolaemia or pain
a HR > 90/min may be the equivalent of a younger adult's HR being > 140/min in risk terms
this is especially problematic if on beta adrenergic blockers which mask the physiologic response to the shocked hypotensive patient and increase mortality - consider using serum lactate and base deficit as markers for occult hypoperfusion
slow bleeding haemothorax may not become clinically evident for days - require close monitoring with serial CXRs or CT to detect in patients with rib fractures
avoid morphine and use fentanyl instead as shorter acting, less toxic metabolites in the renally impaired, and less risk of hypotension due to histamine release
baseline BP may be high so a “normal” BP may be misleading
a systolic BP < 110mmHg instead of below 95mmHg as with younger adults, becomes more concerning as a marker for mortality
delayed onset of clinical symptoms and signs of subdural hematoma
brain reduces in size by around 30% by age 70yrs leaving extra space for haematoma to occupy
peritonism not as detectable
abdominal wall musculature less and pain sensitivity is less